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Showing 1 to 12 of 1279 entries
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Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line.

Toxicology reports

Simon BR, Wilson MJ, Blake DA, Yu H, Wickliffe JK.
PMID: 25170436
Toxicol Rep. 2014 Jul 14;1:391-400. doi: 10.1016/j.toxrep.2014.07.003.

Previously, we demonstrated the sensitivity of RPTEC/TERT1 cells, an immortalized human renal proximal tubule epithelial cell line, to two common environmental carcinogens, cadmium (Cd) and benzo[a]pyrene (B[a]P). Here, we measured BPDE-DNA adducts using a competitive ELISA method after cells...

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Simon BR, Wilson MJ, Blake DA, et al. Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line. Toxicol Rep. 2014;1:391-400doi: 10.1016/j.toxrep.2014.07.003.
Simon, B. R., Wilson, M. J., Blake, D. A., Yu, H., & Wickliffe, J. K. (2014). Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line. Toxicology reports, 1391-400. https://doi.org/10.1016/j.toxrep.2014.07.003
Simon, Bridget R, et al. "Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line." Toxicology reports vol. 1 (2014): 391-400. doi: https://doi.org/10.1016/j.toxrep.2014.07.003
Simon BR, Wilson MJ, Blake DA, Yu H, Wickliffe JK. Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line. Toxicol Rep. 2014 Jul 14;1:391-400. doi: 10.1016/j.toxrep.2014.07.003. PMID: 25170436; PMCID: PMC4142648.
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Bisphenol S impairs blood functions and induces cardiovascular risks in rats.

Toxicology reports

Pal S, Sarkar K, Nath PP, Mondal M, Khatun A, Paul G.
PMID: 29152460
Toxicol Rep. 2017 Oct 20;4:560-565. doi: 10.1016/j.toxrep.2017.10.006. eCollection 2017.

Bisphenol S (BPS) is an industrial chemical which is recently used to replace the potentially toxic Bisphenol A (BPA) in making polycarbonate plastics, epoxy resins and thermal receipt papers. The probable toxic effects of BPS on the functions of...

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Pal S, Sarkar K, Nath PP, et al. Bisphenol S impairs blood functions and induces cardiovascular risks in rats. Toxicol Rep. 2017;4:560-565doi: 10.1016/j.toxrep.2017.10.006.
Pal, S., Sarkar, K., Nath, P. P., Mondal, M., Khatun, A., & Paul, G. (2017). Bisphenol S impairs blood functions and induces cardiovascular risks in rats. Toxicology reports, 4560-565. https://doi.org/10.1016/j.toxrep.2017.10.006
Pal, Sanghamitra, et al. "Bisphenol S impairs blood functions and induces cardiovascular risks in rats." Toxicology reports vol. 4 (2017): 560-565. doi: https://doi.org/10.1016/j.toxrep.2017.10.006
Pal S, Sarkar K, Nath PP, Mondal M, Khatun A, Paul G. Bisphenol S impairs blood functions and induces cardiovascular risks in rats. Toxicol Rep. 2017 Oct 20;4:560-565. doi: 10.1016/j.toxrep.2017.10.006. eCollection 2017. PMID: 29152460; PMCID: PMC5671619.
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Protective role of .

Toxicology reports

Purena R, Seth R, Bhatt R.
PMID: 29487802
Toxicol Rep. 2018 Feb 03;5:270-277. doi: 10.1016/j.toxrep.2018.01.008. eCollection 2018.

Nephrotoxicity is a major limiting factor in cisplatin treatment. In the present study hydro-ethanolic leaf extract of

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Purena R, Seth R, Bhatt R. Protective role of . Toxicol Rep. 2018;5:270-277doi: 10.1016/j.toxrep.2018.01.008.
Purena, R., Seth, R., & Bhatt, R. (2018). Protective role of . Toxicology reports, 5270-277. https://doi.org/10.1016/j.toxrep.2018.01.008
Purena, Rupal, et al. "Protective role of ." Toxicology reports vol. 5 (2018): 270-277. doi: https://doi.org/10.1016/j.toxrep.2018.01.008
Purena R, Seth R, Bhatt R. Protective role of . Toxicol Rep. 2018 Feb 03;5:270-277. doi: 10.1016/j.toxrep.2018.01.008. eCollection 2018. PMID: 29487802; PMCID: PMC5814364.
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Transcriptome-metabolome wide association study (TMWAS) of maneb and paraquat neurotoxicity reveals network level interactions in toxicologic mechanism.

Toxicology reports

Roede JR, Uppal K, Park Y, Tran V, Jones DP.
PMID: 27722094
Toxicol Rep. 2014;1:435-444. doi: 10.1016/j.toxrep.2014.07.006. Epub 2014 Jul 24.

A combination of the herbicide paraquat (PQ) and fungicide maneb (MB) has been linked to Parkinson's disease. Previous studies show that this involves an additive toxicity with at least two different mechanisms. However, detailed understanding of mixtures is often...

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Roede JR, Uppal K, Park Y, et al. Transcriptome-metabolome wide association study (TMWAS) of maneb and paraquat neurotoxicity reveals network level interactions in toxicologic mechanism. Toxicol Rep. 2014;1:435-444doi: 10.1016/j.toxrep.2014.07.006.
Roede, J. R., Uppal, K., Park, Y., Tran, V., & Jones, D. P. (2014). Transcriptome-metabolome wide association study (TMWAS) of maneb and paraquat neurotoxicity reveals network level interactions in toxicologic mechanism. Toxicology reports, 1435-444. https://doi.org/10.1016/j.toxrep.2014.07.006
Roede, James R, et al. "Transcriptome-metabolome wide association study (TMWAS) of maneb and paraquat neurotoxicity reveals network level interactions in toxicologic mechanism." Toxicology reports vol. 1 (2014): 435-444. doi: https://doi.org/10.1016/j.toxrep.2014.07.006
Roede JR, Uppal K, Park Y, Tran V, Jones DP. Transcriptome-metabolome wide association study (TMWAS) of maneb and paraquat neurotoxicity reveals network level interactions in toxicologic mechanism. Toxicol Rep. 2014;1:435-444. doi: 10.1016/j.toxrep.2014.07.006. Epub 2014 Jul 24. PMID: 27722094; PMCID: PMC5053767.
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Use of Toll-like receptor assays for the detection of bacterial contaminations in icodextrin batches released for peritoneal dialysis.

Toxicology reports

Hacine-Gherbi H, Denys A, Carpentier M, Heysen A, Duflot P, Lanos P, Allain F.
PMID: 29152461
Toxicol Rep. 2017 Oct 23;4:566-573. doi: 10.1016/j.toxrep.2017.10.004. eCollection 2017.

Icodextrin is a starch derivative used for preparing solutions of peritoneal dialysis. Unfortunately, peptidoglycans (PGN) and lipopolysaccharides (LPS) have been reported to contaminate certain icodextrin batches and to contribute to the development of sterile peritonitis. The decision of selecting...

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Hacine-Gherbi H, Denys A, Carpentier M, et al. Use of Toll-like receptor assays for the detection of bacterial contaminations in icodextrin batches released for peritoneal dialysis. Toxicol Rep. 2017;4:566-573doi: 10.1016/j.toxrep.2017.10.004.
Hacine-Gherbi, H., Denys, A., Carpentier, M., Heysen, A., Duflot, P., Lanos, P., & Allain, F. (2017). Use of Toll-like receptor assays for the detection of bacterial contaminations in icodextrin batches released for peritoneal dialysis. Toxicology reports, 4566-573. https://doi.org/10.1016/j.toxrep.2017.10.004
Hacine-Gherbi, Hêla, et al. "Use of Toll-like receptor assays for the detection of bacterial contaminations in icodextrin batches released for peritoneal dialysis." Toxicology reports vol. 4 (2017): 566-573. doi: https://doi.org/10.1016/j.toxrep.2017.10.004
Hacine-Gherbi H, Denys A, Carpentier M, Heysen A, Duflot P, Lanos P, Allain F. Use of Toll-like receptor assays for the detection of bacterial contaminations in icodextrin batches released for peritoneal dialysis. Toxicol Rep. 2017 Oct 23;4:566-573. doi: 10.1016/j.toxrep.2017.10.004. eCollection 2017. PMID: 29152461; PMCID: PMC5671617.
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Relationship between self-reported fish and shellfish consumption, carbon and nitrogen stable isotope values and total mercury concentrations in pregnant women (II) from Baja California Sur, Mexico.

Toxicology reports

Bentzen R, Castellini JM, Gaxiola-Robles R, Zenteno-Savín T, Méndez-Rodríguez LC, O'Hara T.
PMID: 28936396
Toxicol Rep. 2014;1:1115-1122. doi: 10.1016/j.toxrep.2014.09.014. Epub 2014 Oct 13.

Seafood is a valuable source of nutrients important for fetal development. However, seafood consumption is the main route of exposure to monomethyl mercury (MeHg

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Bentzen R, Castellini JM, Gaxiola-Robles R, et al. Relationship between self-reported fish and shellfish consumption, carbon and nitrogen stable isotope values and total mercury concentrations in pregnant women (II) from Baja California Sur, Mexico. Toxicol Rep. 2014;1:1115-1122doi: 10.1016/j.toxrep.2014.09.014.
Bentzen, R., Castellini, J. M., Gaxiola-Robles, R., Zenteno-Savín, T., Méndez-Rodríguez, L. C., & O'Hara, T. (2014). Relationship between self-reported fish and shellfish consumption, carbon and nitrogen stable isotope values and total mercury concentrations in pregnant women (II) from Baja California Sur, Mexico. Toxicology reports, 11115-1122. https://doi.org/10.1016/j.toxrep.2014.09.014
Bentzen, Rebecca, et al. "Relationship between self-reported fish and shellfish consumption, carbon and nitrogen stable isotope values and total mercury concentrations in pregnant women (II) from Baja California Sur, Mexico." Toxicology reports vol. 1 (2014): 1115-1122. doi: https://doi.org/10.1016/j.toxrep.2014.09.014
Bentzen R, Castellini JM, Gaxiola-Robles R, Zenteno-Savín T, Méndez-Rodríguez LC, O'Hara T. Relationship between self-reported fish and shellfish consumption, carbon and nitrogen stable isotope values and total mercury concentrations in pregnant women (II) from Baja California Sur, Mexico. Toxicol Rep. 2014;1:1115-1122. doi: 10.1016/j.toxrep.2014.09.014. Epub 2014 Oct 13. PMID: 28936396; PMCID: PMC5598523.
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Sex-Dependent effects of developmental arsenic exposure on methylation capacity and methylation regulation of the glucocorticoid receptor system in the embryonic mouse brain.

Toxicology reports

Allan AM, Hafez AK, Labrecque MT, Solomon ER, Shaikh MN, Zheng X, Ali A.
PMID: 26855884
Toxicol Rep. 2015 Oct;2:1376-1390. doi: 10.1016/j.toxrep.2015.10.003.

Previously we have shown that prenatal moderate arsenic exposure (50 ppb) disrupts glucocorticoid receptor (GR) programming and that these changes continue into adolescence in males. However, it was not clear what the molecular mechanisms were promoting these GR programming...

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Allan AM, Hafez AK, Labrecque MT, et al. Sex-Dependent effects of developmental arsenic exposure on methylation capacity and methylation regulation of the glucocorticoid receptor system in the embryonic mouse brain. Toxicol Rep. 2015;2:1376-1390doi: 10.1016/j.toxrep.2015.10.003.
Allan, A. M., Hafez, A. K., Labrecque, M. T., Solomon, E. R., Shaikh, M. N., Zheng, X., & Ali, A. (2015). Sex-Dependent effects of developmental arsenic exposure on methylation capacity and methylation regulation of the glucocorticoid receptor system in the embryonic mouse brain. Toxicology reports, 21376-1390. https://doi.org/10.1016/j.toxrep.2015.10.003
Allan, Andrea M, et al. "Sex-Dependent effects of developmental arsenic exposure on methylation capacity and methylation regulation of the glucocorticoid receptor system in the embryonic mouse brain." Toxicology reports vol. 2 (2015): 1376-1390. doi: https://doi.org/10.1016/j.toxrep.2015.10.003
Allan AM, Hafez AK, Labrecque MT, Solomon ER, Shaikh MN, Zheng X, Ali A. Sex-Dependent effects of developmental arsenic exposure on methylation capacity and methylation regulation of the glucocorticoid receptor system in the embryonic mouse brain. Toxicol Rep. 2015 Oct;2:1376-1390. doi: 10.1016/j.toxrep.2015.10.003. PMID: 26855884; PMCID: PMC4741109.
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Assessment of the impact of genetically modified LibertyLink.

Toxicology reports

Tyshko NV, Zhminchenko VM, Selyaskin KE, Pashorina VA, Utembaeva NT, Tutelyan VA.
PMID: 28962249
Toxicol Rep. 2014 Jun 11;1:330-340. doi: 10.1016/j.toxrep.2014.05.013. eCollection 2014.

This publication presents the assessment of the impact of genetically modified (GM) LibertyLink

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Tyshko NV, Zhminchenko VM, Selyaskin KE, et al. Assessment of the impact of genetically modified LibertyLink. Toxicol Rep. 2014;1:330-340doi: 10.1016/j.toxrep.2014.05.013.
Tyshko, N. V., Zhminchenko, V. M., Selyaskin, K. E., Pashorina, V. A., Utembaeva, N. T., & Tutelyan, V. A. (2014). Assessment of the impact of genetically modified LibertyLink. Toxicology reports, 1330-340. https://doi.org/10.1016/j.toxrep.2014.05.013
Tyshko, N V, et al. "Assessment of the impact of genetically modified LibertyLink." Toxicology reports vol. 1 (2014): 330-340. doi: https://doi.org/10.1016/j.toxrep.2014.05.013
Tyshko NV, Zhminchenko VM, Selyaskin KE, Pashorina VA, Utembaeva NT, Tutelyan VA. Assessment of the impact of genetically modified LibertyLink. Toxicol Rep. 2014 Jun 11;1:330-340. doi: 10.1016/j.toxrep.2014.05.013. eCollection 2014. PMID: 28962249; PMCID: PMC5598231.
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Survival predictors in paraquat intoxification and role of immunosuppression.

Toxicology reports

Koh KH, Tan CH, Hii LW, Lee J, Ngu LL, Chai AJ, Loh CL, Lam SW, Mushahar L, Fam TL, Yusuf WSM.
PMID: 28962262
Toxicol Rep. 2014 Jul 18;1:490-495. doi: 10.1016/j.toxrep.2014.06.010. eCollection 2014.

Paraquat poisoning resulted in multiorgan failure and is associated with high mortality. We audited 83 historical cases of paraquat poisoning in past 2 years treated with conventional decontamination and supportive treatment, followed by enrolling 85 patients over a 2...

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Koh KH, Tan CH, Hii LW, et al. Survival predictors in paraquat intoxification and role of immunosuppression. Toxicol Rep. 2014;1:490-495doi: 10.1016/j.toxrep.2014.06.010.
Koh, K. H., Tan, C. H., Hii, L. W., Lee, J., Ngu, L. L., Chai, A. J., Loh, C. L., Lam, S. W., Mushahar, L., Fam, T. L., & Yusuf, W. S. M. (2014). Survival predictors in paraquat intoxification and role of immunosuppression. Toxicology reports, 1490-495. https://doi.org/10.1016/j.toxrep.2014.06.010
Koh, Keng-Hee, et al. "Survival predictors in paraquat intoxification and role of immunosuppression." Toxicology reports vol. 1 (2014): 490-495. doi: https://doi.org/10.1016/j.toxrep.2014.06.010
Koh KH, Tan CH, Hii LW, Lee J, Ngu LL, Chai AJ, Loh CL, Lam SW, Mushahar L, Fam TL, Yusuf WSM. Survival predictors in paraquat intoxification and role of immunosuppression. Toxicol Rep. 2014 Jul 18;1:490-495. doi: 10.1016/j.toxrep.2014.06.010. eCollection 2014. PMID: 28962262; PMCID: PMC5598267.
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Effect of livingstonepotato (.

Toxicology reports

Eleazu CO, Eleazu KC, Chukwuma SC, Okoronkwo J, Emelike CU.
PMID: 28962281
Toxicol Rep. 2014 Aug 29;1:674-681. doi: 10.1016/j.toxrep.2014.08.013. eCollection 2014.

The effect of livingstone potato (

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Eleazu CO, Eleazu KC, Chukwuma SC, et al. Effect of livingstonepotato (. Toxicol Rep. 2014;1:674-681doi: 10.1016/j.toxrep.2014.08.013.
Eleazu, C. O., Eleazu, K. C., Chukwuma, S. C., Okoronkwo, J., & Emelike, C. U. (2014). Effect of livingstonepotato (. Toxicology reports, 1674-681. https://doi.org/10.1016/j.toxrep.2014.08.013
Eleazu, C O, et al. "Effect of livingstonepotato (." Toxicology reports vol. 1 (2014): 674-681. doi: https://doi.org/10.1016/j.toxrep.2014.08.013
Eleazu CO, Eleazu KC, Chukwuma SC, Okoronkwo J, Emelike CU. Effect of livingstonepotato (. Toxicol Rep. 2014 Aug 29;1:674-681. doi: 10.1016/j.toxrep.2014.08.013. eCollection 2014. PMID: 28962281; PMCID: PMC5598204.
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[No title available]

Toxicology reports

Pickles J, Rafiq S, Cochrane SA, Lalljie A.
PMID: 28962297
Toxicol Rep. 2014 Oct 16;1:858-870. doi: 10.1016/j.toxrep.2014.10.008. eCollection 2014.

The introduction of novel proteins to food products carries with it the need to assess the potential allergenicity of such materials. Resistance to

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Pickles J, Rafiq S, Cochrane SA, et al. . Toxicol Rep. 2014;1:858-870doi: 10.1016/j.toxrep.2014.10.008.
Pickles, J., Rafiq, S., Cochrane, S. A., & Lalljie, A. (2014). . Toxicology reports, 1858-870. https://doi.org/10.1016/j.toxrep.2014.10.008
Pickles, Juliette, et al. "." Toxicology reports vol. 1 (2014): 858-870. doi: https://doi.org/10.1016/j.toxrep.2014.10.008
Pickles J, Rafiq S, Cochrane SA, Lalljie A. . Toxicol Rep. 2014 Oct 16;1:858-870. doi: 10.1016/j.toxrep.2014.10.008. eCollection 2014. PMID: 28962297; PMCID: PMC5598364.
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Silver nanoparticles do not alter human osteoclastogenesis but induce cellular uptake.

Toxicology reports

Pauksch L, Rohnke M, Schnettler R, Lips KS.
PMID: 28962302
Toxicol Rep. 2014 Oct 18;1:900-908. doi: 10.1016/j.toxrep.2014.10.012. eCollection 2014.

Based on the increasing number of multi-drug resistant bacteria in periprosthetic infections, improvement of the antibacterial activity of commonly used biomaterials must be achieved. The broad-spectrum, high antimicrobial efficacy has made silver nanoparticles a promising new antibacterial agent. However,...

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Pauksch L, Rohnke M, Schnettler R, et al. Silver nanoparticles do not alter human osteoclastogenesis but induce cellular uptake. Toxicol Rep. 2014;1:900-908doi: 10.1016/j.toxrep.2014.10.012.
Pauksch, L., Rohnke, M., Schnettler, R., & Lips, K. S. (2014). Silver nanoparticles do not alter human osteoclastogenesis but induce cellular uptake. Toxicology reports, 1900-908. https://doi.org/10.1016/j.toxrep.2014.10.012
Pauksch, Linda, et al. "Silver nanoparticles do not alter human osteoclastogenesis but induce cellular uptake." Toxicology reports vol. 1 (2014): 900-908. doi: https://doi.org/10.1016/j.toxrep.2014.10.012
Pauksch L, Rohnke M, Schnettler R, Lips KS. Silver nanoparticles do not alter human osteoclastogenesis but induce cellular uptake. Toxicol Rep. 2014 Oct 18;1:900-908. doi: 10.1016/j.toxrep.2014.10.012. eCollection 2014. PMID: 28962302; PMCID: PMC5598169.
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Showing 1 to 12 of 1279 entries